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Tabun
Tabun-2D-skeletal.png
Tabun-3D-balls.png
IUPAC name
Other names GA; Ethyl dimethylphosphoramidocyanidate; Dimethylaminoethoxy-cyanophosphine oxide; Dimethylamidoethoxyphosphoryl cyanide; Ethyl dimethylaminocyanophosphonate; Ethyl ester of dimethylphosphoroamidocyanidic acid; Ethyl phosphorodimethylamidocyanidate; Cyanodimethylaminoethoxyphosphine oxide; Dimethylaminoethodycyanophosphine oxide; EA1205
Properties
Molecular formula C5H11N2O2P
Molar mass 162.13 g mol−1
Appearance Colorless to brown liquid
Density 1.0887 g/cm³ at 25 °C
1.102 g/cm³ at 20 °C
Melting point

-50 °C, 223 K, -58 °F

Boiling point

247.5 °C, 521 K, 478 °F

Solubility in water 9.8 g/100 g at 25 °C
7.2 g/100 g at 20 °C
Vapor pressure 0.07 mmHg (9 Pa)
Hazards
Main hazards Toxic. Fires involving this chemical may result in the formation of hydrogen cyanide
NFPA 704
NFPA 704.svg
2
4
1
 
Flash point 78 °C
Except where noted otherwise, data are given for materials in their standard state (at 25 °C, 100 kPa)
Infobox references

Tabun or GA is an extremely toxic chemical substance. It is a clear, colorless, and tasteless liquid with a faint fruity odor.[1] It is classified as a nerve agent because it fatally interferes with normal functioning of the mammalian nervous system. As a chemical weapon, it is classified as a weapon of mass destruction by the United Nations according to UN Resolution 687, and its production is strictly controlled and stockpiling outlawed by the Chemical Weapons Convention of 1993. Tabun is the first of the so-called G-series nerve agents along with GB (sarin), GD (soman) and GF(cyclosarin).

Although pure tabun is clear, less-pure tabun may be brown. It is a volatile chemical, although less so than either sarin or soman; because of this, tabun can be used to contaminate water.[1]

Tabun can be destroyed with bleaching powder, though the poisonous gas cyanogen chloride is produced.[2]

Contents

Effects of overexposure

The symptoms of exposure include:[2][3][4] nervousness/restlessness, miosis (contraction of the pupil), rhinorrhea (runny nose), excessive salivation, dyspnea (difficulty in breathing due to bronchoconstriction/secretions), sweating, bradycardia (slow heartbeat), loss of consciousness, convulsions, flaccid paralysis, loss of bladder and bowel control, apnea (breathing stopped) and lung blisters. The exact symptoms of overexposure are similar to those created by all nerve agents. Tabun is toxic even in minute doses. The number and severity of symptoms which appear vary according to the amount of the agent absorbed and rate of entry of it into the body. Very small skin dosages sometimes cause local sweating and tremors accompanied with characteristically constricted pupils with few other effects. Tabun is about half as toxic as sarin by inhalation, but in very low concentrations it is more irritating to the eyes than sarin. Also, tabun breaks down slowly, which after repeated exposure can lead to build up in the body.[1]

The effects of tabun appear slowly when tabun is absorbed through the skin rather than inhaled. A victim may absorb a lethal dose quickly, although death may be delayed for one to two hours.[3] It should be noted that a person's clothing can release the toxic chemical for up to 30 minutes after exposure.[1] Inhaled lethal dosages kill in one to ten minutes, and liquid absorbed through the eyes kills almost as fast. However, people who experience mild to moderate exposure to tabun can recover completely, if treated almost as soon as exposure occurred.[1] The LCt50 for tabun is about 400 mg-min/m3[5]

Treatment for suspected tabun poisoning is often three injections of a nerve agent antidote, such as atropine.[4] Pralidoxime chloride (2-PAM Cl) also works as an antidote; however, it must be administered within minutes to a few hours following exposure to be effective.[6]

History

For an in-depth discussion, see main article on nerve agent history

Tabun was the first nerve agent to be discovered by accident in January 1936[1][2][7][8][9] by the German researcher Gerhard Schrader.[9] Schrader was experimenting with a class of compounds called organophosphates, which kill insects by interrupting their nervous systems, in order to create a more effective insecticide for IG Farben, a German chemical and pharmaceutical industry conglomerate, at Elberfield. Instead of a new insecticide, he discovered tabun, a chemical enormously toxic to humans as well as insects.

During World War II, as part of the Grün 3 program, a plant for the manufacture of tabun was established at Dyhernfurth[9] (now Brzeg Dolny, Poland), in 1939. Run by Anorgana, GmbH, the plant finally began production of the substance in 1942.[9] The reason that the plant took so long to get started was the extreme precautions used by the plant.[9] Intermediate products of tabun were corrosive, and had to be contained in quartz or silver-lined vessels. Tabun itself was also highly toxic, and final reactions were conducted behind double glass walls.[9] Large scale manufacturing of the agent resulted in problems with tabun's degradation over time, and only around 12,500 tons of material were manufactured before the plant was seized by the Soviet Army. The plant initially produced shells and aerial bombs using a 95:5 mix of tabun and chlorobenzene, designated "Variant A", and in the latter half of the war switched to "Variant B," a 80:20 mix of tabun and chlorobenzene designed for easier dispersion. The Soviets dismantled the plant and shipped it to Russia.

The United States once had a tabun production program,[4] which ended many decades ago. Like the other Allied governments, the Soviets soon abandoned GA for GB and GD. Large quantities of the German-manufactured agent were dumped into the sea to neutralize the substance.

Since GA is much easier to produce than the other G-series weapons and the process is comparatively widely understood, countries that develop a nerve agent capability but lack advanced industrial facilities often start by producing GA.

In his 1970s-1980s tracts to the medias, U.S. conspiracy theorist Francis E. Dec often claimed the use of tabun (misspelled "tabin")[10] as a covert assassination tool in the U.S.

During the Iran–Iraq War, Iraq employed large quantities of chemical weapons against Iranian ground forces. Although the most commonly used agents were mustard gas and sarin, tabun and cyclosarin were also used.[4][11]

See also

References

  1. ^ a b c d e f Facts About Tabun, National Terror Alert Response System
  2. ^ a b c "Nerve Agent: GA". Cbwinfo.com. http://www.cbwinfo.com/Chemical/Nerve/GA.shtml. Retrieved 2008-11-06.  
  3. ^ a b "Chemical Warfare Weapons Fact Sheets — Tabun — GA Nerve Agent". Usmilitary.about.com. http://usmilitary.about.com/library/milinfo/blchemical-3.htm. Retrieved 2008-11-06.  
  4. ^ a b c d http://www.encyclopedia.com/doc/1G2-3403300733.html
  5. ^ "ATSDR — MMG: Nerve Agents: Tabun (GA); Sarin (GB); Soman (GD); and VX". Atsdr.cdc.gov. http://www.atsdr.cdc.gov/MHMI/mmg166.html. Retrieved 2008-11-06.  
  6. ^ Emergency Response Safety and Health Database. TABUN (GA): Nerve Agent. National Institute for Occupatinal Safety and Health. Accessed April 30, 2009.
  7. ^ Chemical Warfare Weapons Fact Sheets, about.com
  8. ^ Chemical Weapons: Nerve Agents, University of Washington
  9. ^ a b c d e f "A Short History of the Development of Nerve Gases". Noblis.org. http://www.noblis.org/AShortHistoryOfTheDevelopmentOfNerveGases.htm. Retrieved 2008-11-06.  
  10. ^ Branting, Peter (2008). The Dectionary, "Deadly Touch Tabin Needle, The" — A list explaining the recurring concepts found in Dec's flyers.
  11. ^ http://abcnews.go.com/US/story?id=90722&page=1

Additional reading

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